| DOI | Trouver le DOI : https://doi.org/10.1189/jlb.2RU0814-388R |
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| Auteur | Rechercher : Wang, Xiaofeng; Rechercher : Kulka, Marianna1 |
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| Affiliation | - Conseil national de recherches du Canada. Institut national de nanotechnologie
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| Format | Texte, Article |
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| Sujet | arachidonic acid; carboxypeptidase A; chymase; docosahexaenoic acid; docosapentaenoic acid; heparin; histamine; icosanoid; icosapentaenoic acid; interleukin 10; interleukin 13; interleukin 17; interleukin 1beta; interleukin 33; interleukin 4; interleukin 5; interleukin 6; interleukin 8; leukotriene B4; leukotriene C4; leukotriene D4; linolenic acid; major basic protein; omega 3 fatty acid; prostaglandin D2; prostaglandin E; serotonin; thrombocyte activating factor; tryptase; tumor necrosis factor; bone marrow derived mast cell; cell activation; cell adhesion; cell migration; cell viability; cytokine production; gene expression; inflammation; lipid raft; mast cell; mast cell degranulation; mediator release; phagocytosis; signal transduction |
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| Résumé | Mast cells are known to play a vital role in the development of inflammation in allergic responses. Recent studies have indicated that mast cell activation could be modulated by n-3 PUFAs, which have a wide range of well-documented health benefits. In our review, we summarize the recent findings and potential mechanisms of the effect of n-3 PUFAs on mast cell activation. This knowledge could provide new strategies for the development of therapeutic interventions for diseases mediated by mast cells. |
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| Date de publication | 2015-05 |
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| Dans | |
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| Langue | anglais |
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| Publications évaluées par des pairs | Oui |
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| Numéro NPARC | 21275622 |
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| Exporter la notice | Exporter en format RIS |
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| Signaler une correction | Signaler une correction (s'ouvre dans un nouvel onglet) |
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| Identificateur de l’enregistrement | 14c05523-1e2f-4ac3-964a-824723df7e05 |
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| Enregistrement créé | 2015-07-14 |
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| Enregistrement modifié | 2020-04-22 |
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