N-3 polyunsaturated fatty acids inhibit IFN-γ-induced IL-18 binding protein production by prostate cancer cells

From National Research Council Canada

DOIResolve DOI: https://doi.org/10.1007/s00262-014-1630-z
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  1. National Research Council of Canada. Aquatic and Crop Resource Development
  2. National Research Council of Canada. National Institute for Nanotechnology
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Subjectgamma interferon; gamma interferon receptor; interleukin 18 binding protein; Janus kinase 1; messenger RNA; mitogen activated protein kinase 1; omega 3 fatty acid; STAT1 protein; synaptophysin; gamma interferon; gamma interferon receptor; interferon receptor; interleukin-18 binding protein; messenger RNA; signal peptide; cancer cell; cytokine production; immune response; prostate cancer; protein binding; protein expression; protein phosphorylation; signal transduction; biosynthesis; drug effects; gene expression regulation; genetics; metabolism; prostate tumor; tumor cell line; Gene Expression Regulation, Neoplastic; Intercellular Signaling Peptides and Proteins; Prostatic Neoplasms
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NPARC number21275462
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Record identifiere414a4f8-fc9f-40e4-9cbf-ae7d2efc2c84
Record created2015-07-14
Record modified2020-04-22
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