N-3 polyunsaturated fatty acids inhibit IFN-γ-induced IL-18 binding protein production by prostate cancer cells

From National Research Council Canada

DOIResolve DOI: https://doi.org/10.1007/s00262-014-1630-z
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Name affiliation
  1. National Research Council of Canada. Aquatic and Crop Resource Development
  2. National Research Council of Canada. National Institute for Nanotechnology
FormatText, Article
Journal titleCancer Immunology, Immunotherapy
Subjectgamma interferon; gamma interferon receptor; interleukin 18 binding protein; Janus kinase 1; messenger RNA; mitogen activated protein kinase 1; omega 3 fatty acid; STAT1 protein; synaptophysin; gamma interferon; gamma interferon receptor; interferon receptor; interleukin-18 binding protein; messenger RNA; signal peptide; cancer cell; cytokine production; immune response; prostate cancer; protein binding; protein expression; protein phosphorylation; signal transduction; biosynthesis; drug effects; gene expression regulation; genetics; metabolism; prostate tumor; tumor cell line; Gene Expression Regulation, Neoplastic; Intercellular Signaling Peptides and Proteins; Prostatic Neoplasms
Publication date
PublisherSpringer International Publishing
Peer reviewedYes
NPARC number21275462
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Record identifiere414a4f8-fc9f-40e4-9cbf-ae7d2efc2c84
Record created2015-07-14
Record modified2020-04-22
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