Specific inhibition by hGRB10zeta of insulin-induced glycogen synthase activation: evidence for a novel signaling pathway

From National Research Council Canada

DOIResolve DOI: https://doi.org/10.1016/S0303-7207(00)00439-1
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  1. National Research Council of Canada. NRC Biotechnology Research Institute
FormatText, Article
Subjectpharmaceutical; insulin; hGrb10ζ; hepatocytes; PI3-kinase; glycogen synthase; GSK-3; Akt/PKB; 1-phosphatidylinositol 3-kinase; calcium-calmodulin-dependent protein kinases; cells, cultured; enzyme activation; glycogen; glycogen synthase kinase 3; GRB10 adaptor protein; Insulin-like growth factor binding protein 1; male; mitogen-activated protein kinases; organometallic compounds; phenanthrolines; phosphorylation; protein tyrosine phosphatases; protein-serine-threonine kinases; protein; proto-Oncogene Proteins; proto-Oncogene Proteins c-akt; rat; rats, sprague-dawley; receptor, insulin; RNA, messenger; signal transduction; transcription, genetic
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NRC number44778
NPARC number3539576
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Record identifier612aba11-a4df-4de6-96b8-5015199b0cd5
Record created2009-03-01
Record modified2020-03-27
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